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The possible mechanism is that NNMT participates in regulating cell cycle progression via many pathways, such as the p27 and PI3KAkt pathways, enhancing tumorigenesis capacity

Upon exposure to GSH, redox-driven or binding-induced structural changes modulate their emission, enabling highly sensitive and selective GSH quantification with minimal interference from other thiols such as cysteine and homocysteine [93]

USA 98 (8), 46114616 (2001)

NPC-like glioma cells relied on amino acid metabolism and high mitochondrial oxygen consumption

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